Markov Chain Resonances and ChIPSeeker: an R/Bioconductor package for P-level annotation, comparison and visualization
Yu, G., Wang, L.-G. & He, Q.-Y. ChIPseeker: an R/Bioconductor package for ChIP peak annotation, comparison and visualization. 2382–2383 were the results of the Bioinformatics 31, which was published in 2015.
Perovanovic, J. et al. Oct1 is associated with the Smad family of transcription factors. Sci. Signal. 16, eadd5750 (2023).
Farshidfar, F. et al. Integrative molecular and clinical profiling of acral melanoma links focal amplification of 22q11.21 to metastasis. Nat. Commun. 13, 898 (2022).
Cailleau, R., Olivé, M. & Cruciger, Q. V. Long-term human breast carcinoma cell lines of metastatic origin: preliminary characterization. In VITRO 14, the case was referred to as the “911”.
Source: Targeting GRPR for sex hormone-dependent cancer after loss of E-cadherin
Variance stabilization and normalization for one-color microarray data in Bioinformatics 22, 2547-2553 (2006). The Galaxy: The 2022 update
Johnson, W.E. and Li, C. used empirical Bayes methods to adjusting the effects on expression data. Biostatistics 8, 118–127 (2007).
Motakis, E. S., Nason, G. P., Fryzlewicz, P. & Rutter, G. A. Variance stabilization and normalization for one-color microarray data using a data-driven multiscale approach. Bioinformatics 22, 2547–2553 (2006).
The community is called The GALAXY. The Galaxy platform for accessible, reproducible and collaborative biomedical analyses: 2022 update. Nucleic Acids Res. 50, W345–W351 (2022).
Langmead, B., Wilks, C., Antonescu, V. & Charles, R. Scaling read aligners to hundreds of threads on general-purpose processors. Bioinformatics 35, 421–432 (2019).
Source: Targeting GRPR for sex hormone-dependent cancer after loss of E-cadherin
Competition of ligand-activated and YAP enhancers regulates whole-genome estrogen receptor binding in mouse uterine tissue
Hewitt, S. C. et al. The whole-genome estrogenreceptor binding in mouse uterine tissue is a research resource. Mol. The article was published in the journal of acheticollol.
Park, J.-S. et al. Six2 and Wnt regulate self-renewal and commitment of nephron progenitors through shared gene regulatory networks. Dev. Cell 23, 637–651 (2012).
Rhie, S. K. et al. Identification of activated enhancers and linked transcription factors in breast, prostate, and kidney tumors by tracing enhancer networks using epigenetic traits. Epigenetics Chromatin 9, 50 (2016).
Nair, S. J. et al. Phase separation of ligand-activated enhancers licenses cooperative chromosomal enhancer assembly. Nat. Struct. Mol. Biol. 26, 193–203 (2019).
SMADs and YAP compete for control of the elongation of -catenin:leF-1-recruited mRNAPII. Mol. Cell 58, 780–793 (2015).
Source: Targeting GRPR for sex hormone-dependent cancer after loss of E-cadherin
The 26th Bioinformatics Report on p16INK4a Recombination in the C57BL/6 Mice NRASQ61K melanoma Cell Lineage
Petit et al. C57BL/6 congenic mouse NRASQ61K melanoma cell lines are highly sensitive to the combination of Mek and Akt inhibitors in vitro and in vivo. Pigment Cell Melanoma Res. 32, 829–841 (2019).
Robinson, M. D., McCarthy, D. J. & Smyth, G. K. edgeR: a Bioconductor package for differential expression analysis of digital gene expression data. The 26th Bioinformatics report was published in 2010.
Leek, J. T., Johnson, W. E., Parker, H. S., Jaffe, A. E. & Storey, J. D. The sva package for removing batch effects and other unwanted variation in high-throughput experiments. A new book on the subject of Bioinformatics was published in 2012
Delmas, V., Martinozzi, S., Bourgeois, Y., Holzenberger, M. & Larue, L. Cre-mediated recombination in the skin melanocyte lineage. Genesis 36, 73–80 was published in 2003
Berx, G. & Van Roy, F. The E-cadherin/catenin complex: an important gatekeeper in breast cancer tumorigenesis and malignant progression. Breast Cancer Res. 3 was published in 2001.
V. Delmas et al. β-Catenin induces immortalization of melanocytes by suppressing p16INK4a expression and cooperates with N-Ras in melanoma development. Genes Dev. 21, 2923–2935 (2007).
Source: Targeting GRPR for sex hormone-dependent cancer after loss of E-cadherin
gganatogram: A modular R package for modular visualisation of anatograms and tissues based on ggplot2
Maag, J. L. V. gganatogram: An R package for modular visualisation of anatograms and tissues based on ggplot2. F 1000 Research by F 1000 was published in June of 2018?
Mills, K. R., Misra, J. & Torabifard, H. Allosteric modulation of the YAP/TAZ-TEAD interaction by palmitoylation and small-molecule inhibitors. J. Phys. Chem. B 128, 3795–3806 will be in the future.
He, Z., Li, R., and Jiang,H. studied the Hippo pathway components in human cancers. Front. Cell Dev. There is an article in the journal of Biol. 9, 661718.
Source: Targeting GRPR for sex hormone-dependent cancer after loss of E-cadherin
Gastrin receptors for cancer and itch therapy. Phase I trial of the bombesin/gastrin-releasing peptide antagonist RC3095
Peng, S. et al. Structures of human gastrin-releasing peptide receptors bound to antagonist and agonist for cancer and itch therapy. Proc. There is a Natl Acad. USA 120, e221634060.
L. L. et al. Loss of bombesin-induced feeding suppression in gastrin-releasing peptide receptor-deficient mice. There was a Proc. It’s Natl Acad. USA 95, 3188–2 was published in 1998.
Schwartsmann, G. et al. A phase I trial of the bombesin/gastrin-releasing peptide (BN/GRP) antagonist RC3095 in patients with advanced solid malignancies. Invest. New drugs 24, 403–412 were released.
Source: Targeting GRPR for sex hormone-dependent cancer after loss of E-cadherin
Analgesic effect of gastrin-releasing peptide in the dorsal horn: a study of estrogens for treatment of hormone-dependent diseases
Polgár, E. et al. A population of superficial horn vertical cells plays a part in both itch and pain. Pain 164, 149–170 (2023).
Saeki, A., Yamanaka, H., Kobayashi, K., Okubo, M. & Noguchi, K. Analgesic effect of gastrin-releasing peptide in the dorsal horn. It is also called mos. Pain 18, 17448069221108965 (2022).
Rižner, T. L. & Romano, A. Targeting the formation of estrogens for treatment of hormone dependent diseases–current status. Front. 13, 1155558 (2023).
Source: Targeting GRPR for sex hormone-dependent cancer after loss of E-cadherin
Activation of receptor tyrosine kinases in a type of breast cancer after loss of E-cadherin
Hsiao, P.-W., Fryer, C. J., Trotter, K. W., Wang, W. & Archer, T. K. BAF60a mediates critical interactions between nuclear receptors and the BRG1 chromatin-remodeling complex for transactivation. There is a name for it. The cell. There were 23 articles in the Biol. 23, 6210–6 220.
J., Soares, L. M., Kadoch, C. and Chan, H. M. Trends Genet. In 2020, there is 36, 936–950.
A. et al. It has been found that the loss of E-cadherin causes the creation of a pathway in a type of breast cancer. The name is named after a person. Cancer Res. 20, 1405–1419 (2022).
Qian, X., Karpova, T., Sheppard, A. M., McNally, J. & Lowy, D. R. E-cadherin-mediated adhesion inhibits ligand-dependent activation of diverse receptor tyrosine kinases. EMBO J. 23, 1739–1748 (2004).
O. G., Morali and others. IGF-II induces rapid β-catenin relocation to the nucleus during epithelium to mesenchyme transition. Oncogene 20, 4942–4950 (2001).
Source: Targeting GRPR for sex hormone-dependent cancer after loss of E-cadherin
Determination of estrogenic potential in waste water without sample extraction: a new method for identifying genetic alterations associated with sex hormone-dependent cancer after loss of E-cadherin
Avberšek, M., Žegura, B., Filipič, M., Uranjek-Ževart, N. & Heath, E. Determination of estrogenic potential in waste water without sample extraction. J. Hazard. 260, 556, and 533 were published.
Venza, M. Clinicopathological features of melanoma can be linked to the genetic alterations associated with E-cadherin. Oncol. Rep. 35 was enacted in the year 2016
H., and coworkers, written by Sung. Global cancer statistics in 2020. GLOBOCAN estimates of incidence and mortality worldwide for 36 cancers in 185 countries. CA Cancer J. Clin. 71 will be published in 2021.
Source: Targeting GRPR for sex hormone-dependent cancer after loss of E-cadherin
Evidence for sex hormone-dependent cancer in tall women treated with high-dose oestrogen during adolescence
Cano, A. et al. The transcription factor Snail contributes to the control of the transitions in the body. Nat. Cell Biol. 2, 76–83 was written in 2000.
Y. Y. Lee, et al. Incidence and outcomes of pregnancy-associated cancer in Australia, 1994–2008: a population-based linkage study. The BJOG took place in the year of 2012
E. Benyi and others, et al. Risks of malignant and non-malignant tumours in tall women treated with high-dose oestrogen during adolescence. It’s Horm. There is a Res. Paediatr. 82, 89–96 (2014).
Source: Targeting GRPR for sex hormone-dependent cancer after loss of E-cadherin
De Novo Fatty Acid Synthesis in Neuronal Tissue: a Molecular Target for Treatment of Femtosis and Melanoma
J. Ackermann is the author of this article. The emergence of melanoma can be traced back to the presence of activated N- Ras Q61K on an INK4a deficient background. The Cancer Res 65 describes the consequences of cancer.
Mashima, T., Seimiya, H. & Tsuruo, T. De novo fatty-acid synthesis and related pathways as molecular targets for cancer therapy. Br. J. Cancer 100, 1369–1372 (2009).
Medes, G., Thomas, A. & Weinhouse, S. Metabolism of neoplastic tissue. IV. A study of lipid synthesis in neoplastic tissue slices in vitro. Cancer Res. 13 was published in1953.
Lang, X. Radiotherapy and immunotherapy promote tumoral lipid oxidation and ferroptosis via synergistic repression of SLC7A11. Cancer Discov. 9, 1695–1695.
Cai, Y. et al. The Nrf2/HOT-1 pathway is activated by vitamins D and D3. Transl. Pediatr. 11, 1633–1644 (2022).
Source: Glycosaminoglycan-driven lipoprotein uptake protects tumours from ferroptosis
The Role of Glycosaminoglycan-Driven Lipoprotein Uptake Protects Cancer Cells from Ferroptosis
Hoofnagle, A. N. & Heinecke, J. W. Lipoproteomics: using mass spectrometry-based proteomics to explore the assembly, structure, and function of lipoproteins. J.Lonic Res. 50 was published in 1967.
Shah, Farmer and Zilka were involved in the study of how autoxidation is prevented by the use of fluorescence-enabled drugs. Cell Chem. Biol. 26, 1594–1607 (2019).
W. Ou, and others. Low-density lipoprotein docosahexaenoic acid nanoparticles induce ferroptotic cell death in hepatocellular carcinoma. Free Radic. The journal of biology. Med. 112, 597–607 (2017).
The antiporter xCT enhances cancer cell dependency onglucose by exporting glutamate. The J. Biol. is an inscrutable journal. Chem. 292, 14 243, 146 and 149.
Asimakopoulou, A. P., Theocharis, A. D., Tzanakakis, G. N. & Karamanos, N. K. The role of chondroitin sulfate in cancer and other diseases. The book is called In Vivo 22, 385–389.
synthetic lethal screening identifies compounds that cause iron dependent and nonapoptotic cell death in cancer cells That’s Chem. It was vol 15, 234– 25.
Source: Glycosaminoglycan-driven lipoprotein uptake protects tumours from ferroptosis
Cellular dynamics and cytoprotection by glycosaminoglycan-driven lipoprotein uptake protects tumours from ferroptosis
Basu, A., Patel, N. G., Nicholson, E. D. & Weiss, R. J. Spatiotemporal diversity and regulation of glycosaminoglycans in cell homeostasis and human disease. Am. It is the topic of the journal J. Physiol. Cell Physiol. 322, C849–C864.
Matsuda, M. There is a need for SREAPPS to increase the amount ofcholesterol in the liver caused by cholesterol deprivation. The 19th edition of the Genes Dev. 15, 1205–1216 was published in 2001.
Birsoy, K. et al. The electron transport chain has an important role to play in cell proliferation. Cell 161, 540–555.
The low density lipoproteins are released from the cell surface by sulfated sugars. Cell 7, 85–95 (1976).
Klaassen, C. D. & Boles, J. W. Sulfation and sulfotransferases 5: the importance of 3′-phosphoadenosine 5′-phosphosulfate (PAPS) in the regulation of sulfation. FASEB J. 11 was published in 1997.
C., Westley, J., and Schwartz, N.B. are authors. Chemical 30, 7477–483.
The Bulletin of the Society of Engineering Engineers, O. et al. On the mechanism of cytoprotection by ferrostatin-1 and liproxstatin-1 and the role of lipid peroxidation in ferroptotic cell death. The amount of cash of the company is referred to as the ACS Cent. Sci. 3, 232–243 (2017).
Source: Glycosaminoglycan-driven lipoprotein uptake protects tumours from ferroptosis
The Versican-Hyaluronan Complex of a Polyaminoglycan-Driven Lipoprotein in Hematoendothelial Progenitors
Pownall, H. J., Rosales, C., Gillard, B. K. & Gotto, A. M. High-density lipoproteins, reverse cholesterol transport and atherogenesis. Nat. Rev. Cardiol. 18, 712–723 (2021).
Kurokawa, S., Bellinger, F. P., Hill, K. E., Burk, R. F. & Berry, M. J. Isoform-specific binding of selenoprotein P to the β-propeller domain of apolipoprotein E receptor 2 mediates selenium supply. The J. Biol. is about biology Chem. 289, 9195–9207 (2014).
S. and Nandadasa were involved in the project. The versican-hyaluronan complex provides an essential extracellular matrix niche for Flk1+ hematoendothelial progenitors. The matrix was 40–57 during the year.
Source: Glycosaminoglycan-driven lipoprotein uptake protects tumours from ferroptosis
Glucosaminoglycan-driven protein uptake protects breast cancer from ferroptosis: A meta-analysis of the Clear Cell Renal Cell Cancer
Harris, F. M., Best, K. B. & Bell, J. D. Use of laurdan fluorescence intensity and polarization to distinguish between changes in membrane fluidity and phospholipid order. Biochim. Biophys. Acta 1565, 123–128 (2002).
The elimination of breast tumors caused by chondroitin sulfate promoted the growth of new tumors. Genet. Mol. Res. 10 was published in 2011.
X., Li, Q., Che, X., Wang, Q. There is a summary of the process and abnormality of the metabolisms in Clear cell renal cell carcinoma. Front. In Oncol, 11, 727778.
Source: Glycosaminoglycan-driven lipoprotein uptake protects tumours from ferroptosis
Glucosaminoglycan-driven lipoprotein uptake protects against ferroptosis in human monocyte-derived macrophages
Klein, E. A. Vitamin E and the risk of prostate cancer: The Selenium and Vitamin E Cancer Prevention Trial (SELECT). JAMA 306, 1549–1556 (2011).
Hurt, E., Bondjers, G. & Camejo, G. Interaction of LDL with human arterial proteoglycans stimulates its uptake by human monocyte-derived macrophages. J. Lipid Res. 31, 443–454 (1990).
Brandes, C. Alternative splicing in the ligand binding domain of mouse ApoE receptor-2 produces receptor variants binding reelin but not α2-macroglobulin. A.J. Biol. Chem. 276, 22160–22169 (2001).
Source: Glycosaminoglycan-driven lipoprotein uptake protects tumours from ferroptosis
A new quantitative tool for analyzing ubiquinone via liquid chromatography and electron spraying tandem mass spectrometry: Application to serum alpha-tocopherol (vitamin E)
Garcia-Bermudez, J. et al. Stimulation of macropinocytosis overcomes the limitation in cancer cells. The Nat. It was a Metab. 4, 712–738.
Guo, L. S., Hamilton, R. L., Goerke, J., Weinstein, J. N. & Havel, R. J. Interaction of unilamellar liposomes with serum lipoproteins and apolipoproteins. J. Lipid Res. was published in 1980.
The database provides a broad chemical and biological resource. Nat. Methods 17 is for 2020.
De Leenheer, A. P., De Bevere, V. O., Cruyl, A. A. & Claeys, A. E. Determination of serum alpha-tocopherol (vitamin E) by high-performance liquid chromatography. They are looking for a person that knows how to handle Clin. Chem. 24, 585–590 was published in 1978.
In order to quantitate ubiquinone (coenzyme Q10) in a sample, it is necessary to use liquid chromatography electron spraying tandem mass spectrometry. Methods Mol. Biol. 1378, 61–69 (2016).
A. Basu, et al. Quantitative HILIC-Q-TOF-MS analysis of glycosaminoglycans and non-reducing end carbohydrate biomarkers via glycan reductive isotopic labeling. Preprint at ChemRxiv https://doi.org/10.26434/chemrxiv-2025-60p82 (2025).
Source: Glycosaminoglycan-driven lipoprotein uptake protects tumours from ferroptosis
A Mouse Model with a High Barcode Diversity for Molecule Data Analysis of Single Cells. Fat 186, 5183-5999 (2023)
Li, L. et al. A mouse model with high clonal barcode diversity can be used to profile single cells. Cell 186, 5183–5199 (2023).
A platform for drug testing based on human cancer tumors. Nat. The Protoc happened in the 19th century.
FAIR principles for mass spectrometry data were realized by GlycoPOST. D 1523 and D1528 are the nucleic Acids Res. 49.